Concomitant AID Expression and BCL7A Loss Associates With Accelerated Phase Progression and Imatinib Resistance in Chronic Myeloid Leukemia-Reference-Cited by-同舟云学术

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Concomitant AID Expression and BCL7A Loss Associates With Accelerated Phase Progression and Imatinib Resistance in Chronic Myeloid Leukemia

Published:2017-03-01 Issue:2 Volume:37 Page:177-179
ISSN:2234-3806
Container-title:Annals of Laboratory Medicine
language:en
Short-container-title:Ann Lab Med

Author:

Yu Nae¹,Shin Saeam²,Choi Jong Rak¹,Kim Yoonjung³,Lee Kyung-A¹

Affiliation:

1. Department of Laboratory Medicine, Yonsei University College of Medicine, Seoul, Korea

2. Department of Laboratory Medicine, Hallym University College of Medicine, Kangnam Sacred Heart Hospital, Seoul, Korea

3. Department of Laboratory Medicine, Yonsei University Wonju College of Medicine, Wonju, Korea

Funder

Ministry of Health and Welfare

Publisher

Annals of Laboratory Medicine

Subject

Biochemistry, medical,Clinical Biochemistry,General Medicine

Link

https://synapse.koreamed.org/pdf/10.3343/alm.2017.37.2.177

Reference10 articles.

1. Imatinib Resistance and Progression of CML to Blast Crisis: Somatic Hypermutation AIDing the Way

2. Genetic Events Other than BCR-ABL1

3. The B Cell Mutator AID Promotes B Lymphoid Blast Crisis and Drug Resistance in Chronic Myeloid Leukemia

4. Activation-induced cytidine deaminase acts as a mutator in BCR-ABL1–transformed acute lymphoblastic leukemia cells

5. Wnt signaling induces transcription, spatial proximity, and translocation of fusion gene partners in human hematopoietic cells

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4. MiR-501-3p promotes osteosarcoma cell proliferation, migration and invasion by targeting BCL7A;Human Cell;2021-01-07

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