Treatment With Bone Morphogenetic Protein 2 Limits Infarct Size After Myocardial Infarction in Mice-Reference-Cited by-同舟云学术

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Treatment With Bone Morphogenetic Protein 2 Limits Infarct Size After Myocardial Infarction in Mice

Published:2013-04 Issue:4 Volume:39 Page:353-360
ISSN:1073-2322
Container-title:Shock
language:en
Short-container-title:

Author:

Ebelt Henning,Hillebrand Ina,Arlt Stephan,Zhang Ying,Kostin Sawa,Neuhaus Herbert,Müller-Werdan Ursula,Schwarz Elisabeth,Werdan Karl,Braun Thomas

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Critical Care and Intensive Care Medicine,Emergency Medicine

Reference36 articles.

1. : Intracoronary cardiosphere-derived cells for heart regeneration after myocardial infarction (CADUCEUS): a prospective, randomised phase 1 trial.;Makkar;Lancet,2012

2. A role for bone morphogenetic proteins in the induction of cardiac myogenesis.;Schultheiss;Genes Dev,1997

3. Bone morphogenetic protein signaling modulates myocardin transactivation of cardiac genes.;Callis;Circ Res,2005

4. : p38 Mitogen-activated protein kinase controls a switch between cardiomyocyte and neuronal commitment of murine embryonic stem cells by activating myocyte enhancer factor 2C-dependent bone morphogenetic protein 2 transcription.;Wu;Stem Cells Dev,2010

5. Mice deficient for BMP-2 are nonviable and have defects in amnionchorion and cardiac development.;Zhang;Development,1996

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