UnCRMPing Nav1.7 to treat trigeminal neuropathic pain-Reference-Cited by-同舟云学术

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UnCRMPing Nav1.7 to treat trigeminal neuropathic pain

Published:2023-09-06 Issue: Volume: Page:
ISSN:0304-3959
Container-title:Pain
language:en
Short-container-title:PAIN

Author:

Antunes Flavia T.T.¹²,Zamponi Gerald W.¹²

Affiliation:

1. Department of Clinical Neurosciences, Cumming School of Medicine, Alberta Children's Hospital Research Institute, University of Calgary, Calgary, AB, Canada

2. Cumming School of Medicine, Hotchkiss Brain Institute, University of Calgary, Calgary, AB, Canada

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Anesthesiology and Pain Medicine,Neurology (clinical),Neurology

Reference20 articles.

1. Electrophysiological characterization of a Cav3.1 calcium channel mutation linked to trigeminal neuralgia;Alaklabi;Pflugers Arch,2023

2. Painful and painless mutations of SCN9A and SCN11A voltage-gated sodium channels;Baker;Pflugers Arch,2020

3. European Academy of Neurology guideline on trigeminal neuralgia;Bendtsen;Eur J Neurol,2019

4. Small molecule targeting NaV1.7 via inhibition of the CRMP2-Ubc9 interaction reduces and prevents pain chronification in a mouse model of oxaliplatin-induced neuropathic pain;Braden;Neurobiol Pain,2022

5. Selective targeting of NaV1.7 via inhibition of the CRMP2-Ubc9 interaction reduces pain in rodents;Cai;Sci Transl Med,2021

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