Does acute promyelocytic leukemia patient with the STAT5B/RARa fusion gene respond well to decitabine?
Author:
Funder
National Natural Science Foundation of China
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
General Medicine
Reference18 articles.
1. Mechanisms of action and resistance to all-trans retinoic acid (ATRA) and arsenic trioxide (As2O3) in acute promyelocytic leukemia;Tomita;Int J Hematol,2013
2. Acute promyelocytic leukemia: a review and discussion of variant translocations;Adams;Arch Pathol Lab Med,2015
3. The signal transducer and activator of transcription STAT5b gene is a new partner of retinoic acid receptor alpha in acute promyelocytic-like leukaemia;Arnould;Hum Mol Genet,1999
4. All-trans retinoic acid and arsenic trioxide resistance of acute promyelocytic leukemia with the variant STAT5B-RARA fusion gene;Strehl;Leukemia,2013
5. Detection of the STAT5B-RARА fusion transcript in acute promyelocytic leukemia with the normal chromosome 17 on G-banding;Kusakabe;Eur J Haematol,2008
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1. Identification of concurrent STAT3::RARA and RARA::STAT5b fusions in a variant APL case;Molecular Carcinogenesis;2023-12-28
2. Chest Pain Diagnosed as Acute Leukemia: Focus on Coagulation Abnormalities Rather Than White Blood Cell Count;Cureus;2023-03-10
3. Case report: Identification of a novel HNRNPC::RARG fusion in acute promyelocytic leukemia lacking RARA rearrangement;Frontiers in Oncology;2023-01-09
4. Atypical Rearrangements in APL-Like Acute Myeloid Leukemias: Molecular Characterization and Prognosis;Frontiers in Oncology;2022-04-12
5. Treatment of STAT5b-RARA positive acute promyelocytic leukemia by Venetoclax combining with homoharringtonine, cytarabine: A case report and literature review;Blood Science;2022-04
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