Impaired Innate Immune Function Associated with Fecal Supernatant from Crohnʼs Disease Patients-Reference-Cited by-同舟云学术

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Impaired Innate Immune Function Associated with Fecal Supernatant from Crohnʼs Disease Patients

Published:2014-07 Issue:7 Volume:20 Page:1139-1146
ISSN:1078-0998
Container-title:Inflammatory Bowel Diseases
language:en
Short-container-title:Inflammatory Bowel Diseases

Author:

Meelu Parool,Marin Romela,Clish Clary,Zella Garret,Cox Stephen,Yajnik Vijay,Nguyen Deanna D.,Korzenik Joshua R.

Publisher

Oxford University Press (OUP)

Subject

Gastroenterology,Immunology and Allergy

Reference42 articles.

1. Unravelling the pathogenesis of inflammatory bowel disease

2. Genome-wide association defines more than 30 distinct susceptibility loci for Crohn's disease

3. Host–microbe interactions have shaped the genetic architecture of inflammatory bowel disease

4. Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility loci

5. Penetrance of NOD2/CARD15 genetic variants in the general population

Cited by 9 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Fecal Supernatants from Patients with Crohn’s Disease Induce Inflammatory Alterations in M2 Macrophages and Fibroblasts;Cells;2023-12-27

2. Food and Gut Microbiota-Derived Metabolites in Nonalcoholic Fatty Liver Disease;Foods;2022-09-05

3. IL-18 maintains the homeostasis of mucosal immune system via inflammasome-independent but microbiota-dependent manner;Science Bulletin;2021-10

4. Advanced oxidation protein products impair autophagic flux in macrophage by inducing lysosomal dysfunction via activation of PI3K-Akt-mTOR pathway in Crohn's disease;Free Radical Biology and Medicine;2021-08

5. The role of fecal sulfur metabolome in inflammatory bowel diseases;International Journal of Medical Microbiology;2021-07

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