EOSINOPHILIC DROPLET FORMATION, A MORPHOLOGIC MANIFESTATION OF CARDIOMYOCYTE APOPTOSIS, IS PRESENT IN THE HUMORAL (MICROVASCULAR) REJECTION OF HUMAN CARDIAC ALLOGRAFTS
Author:
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Transplantation
Reference13 articles.
1. APOPTOSIS IS THE MAIN MECHANISM OF CARDIOMYOCYTE DEATH IN THE HYPERACUTE REJECTION OF HEART XENO- AND ALLOGRAFTS
2. Correspondence
3. A clinicopathologic study of patients with hemorrhagic myocardial infarction treated with selective coronary thrombolysis with urokinase.
4. A role for secretory phospholipase A2 and C-reactive protein in the removal of injured cells
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1. Further evidence of cardiomyocyte apoptosis in humoral (microvascular) rejection of human cardiac allografts;The Journal of Heart and Lung Transplantation;2004-03
2. Why complement attacks cardiomyocytes in hyperacute heart rejection;Journal of Thoracic and Cardiovascular Surgery;2001-02
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4. MYOCARDIAL DEFECTS IN HYPERACUTE CARDIAC REJECTION;Transplantation;2000-08
5. Why primary angioplasty is less offensive to the myocardium compared with thrombolysis for acute myocardial infarction;American Heart Journal;2000-08
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