APOPTOSIS IS THE MAIN MECHANISM OF CARDIOMYOCYTE DEATH IN THE HYPERACUTE REJECTION OF HEART XENO- AND ALLOGRAFTS
Author:
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Transplantation
Reference18 articles.
1. Eosinophilic droplets similar to red cells are present in the hyperacute rejection of heart xenografts
2. MYOCARDIAL PSEUDOVASCULAR TUBES ARE PRESENT IN THE DELAYED REJECTION OF HEART XENOGRAFTS
3. REPLY: MYOCARDIAL PSEUDOVASCULAR TUBES ARE PRESENT IN THE DELAYED REJECTION OF HEART XENOGRAFTS
4. Letter to the editor: Do neutrophils contribute to the clearance of injured cardiomyocytes in reperfusion injury?
5. Authors' Reply
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1. Ischemic Injury Enhances Dendritic Cell Immunogenicity via TLR4 and NF-κB Activation;The Journal of Immunology;2010-02-17
2. IL-4 and IL-13 Induce Protection of Porcine Endothelial Cells from Killing by Human Complement and from Apoptosis through Activation of a Phosphatidylinositide 3-Kinase/Akt Pathway;The Journal of Immunology;2005-07-20
3. Rejection Severity Directly Correlates With Myocyte Apoptosis in Pig-to-Baboon Cardiac Xenotransplantation;The Journal of Heart and Lung Transplantation;2005-07
4. Cellular Destruction Following Transmyocardial Laser Revascularization (TMR);Journal of Molecular Histology;2005-05
5. Oxidative stress in organ preservation: a multifaceted approach to cardioplegia;Biomedicine & Pharmacotherapy;2005-05
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