Enhanced Platelet Response to Clopidogrel in Abcc3-deficient Mice Due to Its Increased Bioactivation-Reference-Cited by-同舟云学术

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Enhanced Platelet Response to Clopidogrel in Abcc3-deficient Mice Due to Its Increased Bioactivation

Published:2016-12 Issue:6 Volume:68 Page:433-440
ISSN:0160-2446
Container-title:Journal of Cardiovascular Pharmacology
language:en
Short-container-title:

Author:

Tai Ting,Mi Qiong-Yu,Ji Jin-Zi,Yin Qian,Pan Yu-Qin,Zhang Meng-Ran,Huang Bei-Bei,Xie Hong-Guang

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Pharmacology

Reference36 articles.

1. Characterization of MOAT-C and MOAT-D, new members of the MRP/cMOAT subfamily of transporter proteins;Belinsky;J Natl Cancer Inst.,1998

2. MRP3, an organic anion transporter able to transport anti-cancer drugs;Kool;Proc Natl Acad Sci U S A.,1999

3. Tissue distribution and induction of human multidrug resistant protein 3;Scheffer;Lab Invest.,2002

4. Mice lacking Mrp3 (Abcc3) have normal bile salt transport, but altered hepatic transport of endogenous glucuronides;Zelcer;J Hepatol.,2006

5. Increasing systemic exposure of methotrexate by active efflux mediated by multidrug resistance-associated protein 3 (mrp3/abcc3);Kitamura;J Pharmacol Exp Ther.,2008

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3. Short-term standard alcohol consumption enhances platelet response to clopidogrel through inhibition of Nrf2/Ces1 pathway and induction of Cyp2c in mice;Life Sciences;2021-08

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