Functional Modeling of the ACVR1 (R206H) Mutation in FOP
Author:
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Orthopedics and Sports Medicine,General Medicine,Surgery
Reference22 articles.
1. Serrano de la Pena L, Shore EM, Kaplan FS. Paresis of a bone morphogenetic protein-antagonist response in a genetic disorder of heterotopic skeletogenesis.;Ahn;J Bone Joint Surg Am,2003
2. Fibrodysplasia ossificans progressiva (FOP), a disorder of ectopic osteogenesis, misregulates cell surface expression and trafficking of BMPRIA.;de la Pena;J Bone Miner Res,2005
3. Dysregulation of the BMP-p38 MAPK signaling pathway in cells from patients with fibrodysplasia ossificans progressiva (FOP).;Fiori;J Bone Miner Res,2006
4. Discovery of 4-{4-[3-(pyridin-2-yl)-1H-pyrazol-4-yl]pyridin-2-yl}-N-(tetrahydro-2H- pyran-4-yl)benzamide (GW788388): a potent, selective, and orally active transforming growth factor- type I receptor inhibitor.;Gellibert;J Med Chem,2006
5. Treatment considerations for the management of Fibrodysplasia Ossificans Progressiva.;Glaser;Clin Rev Bone Miner Metab,2005
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