ADAM-17 Is Activated by the Mitogenic Protein Kinase ERK in a Model of Kidney Fibrosis

Author:

Navar L. Gabriel,Gooz Monika,Bell Hannah L.,Gööz Monika

Publisher

Elsevier BV

Subject

General Medicine

Reference8 articles.

1. Dual blockade of the renin-angiotensin system with angiotensin-converting enzyme inhibitors and angiotensin II receptor blockers in chronic kidney disease;Song;J Hypertens Suppl,2006

2. Alili R, et al;Lautrette;Angiotensin II and EGF receptor cross-talk in chronic kidney diseases: a new therapeutic approach. nat Med,2005

3. 5-HT2A receptor induces ERK phosphorylation and proliferation through ADAM-17 tumor necrosis factor-alpha-converting enzyme (TACE) activation and heparin-bound epidermal growth factor-like growth factor (HB-EGF) shedding in mes-angial cells;Gooz;J Biol Chem,2006

4. Kozlosky CJ, et al;Black;A metalloproteinase disin-tegrin that releases tumour-necrosis factor-alpha from cells. nature,1997

5. The ADAM gene family: surface proteins with adhesion and protease activity;Primakoff;Trends Genet,2000

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