Expression of hMTH1 in the hippocampi of control and Alzheimer's disease
Author:
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
General Neuroscience
Reference18 articles.
1. Reactive Oxygen Species and the Central Nervous System
2. MutT-related error avoidance mechanism for DNA synthesis
3. Base excision repair of 8-hydroxyguanine protects DNA from endogenous oxidative stress
4. Molecular genetics and structural biology of human MutT homolog, MTH1
5. The Oxidized Forms of dATP Are Substrates for the Human MutT Homologue, the hMTH1 Protein
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3. MUTYH Actively Contributes to Microglial Activation and Impaired Neurogenesis in the Pathogenesis of Alzheimer’s Disease;Oxidative Medicine and Cellular Longevity;2021-12-21
4. Deficiency of MTH1 and/or OGG1 increases the accumulation of 8-oxoguanine in the brain of the AppNL-G-F/NL-G-F knock-in mouse model of Alzheimer’s disease, accompanied by accelerated microgliosis and reduced anxiety-like behavior;Neuroscience Research;2021-11
5. MTH1 and OGG1 maintain a low level of 8-oxoguanine in Alzheimer's brain, and prevent the progression of Alzheimer's pathogenesis;Scientific Reports;2021-03-23
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