SCID Due to ZAP-70 Deficiency
Author:
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Oncology,Hematology,Pediatrics, Perinatology and Child Health
Reference47 articles.
1. Human Severe Combined Immunodeficiency Due to a Defect in ZAP-70, a T Cell Tyrosine Kinase
2. ZAP-70 Deficiency in an Autosomal Recessive form of Severe Combined Immunodeficiency
3. Defective T cell receptor signaling and CD8+ thymic selection in humans lacking Zap-70 kinase
4. Severe Combined Immunodeficiency with Absence of Peripheral Blood CD8+ T Cells Due to ZAP-70 Deficiency
5. Absence of ZAP-70 prevents signaling through the antigen receptor on peripheral blood T cells but not on thymocytes.
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1. A partial human LCK defect causes a T cell immunodeficiency with intestinal inflammation;Journal of Experimental Medicine;2023-11-14
2. Combined immunodeficiency caused by pathogenic variants in the ZAP70 C-terminal SH2 domain;Frontiers in Immunology;2023-05-29
3. Immunodeficiencies;Clinical Immunology;2023
4. T and NK cell lymphoma cell lines do not rely on ZAP-70 for survival;PLOS ONE;2022-01-25
5. Novel Compound Heterozygous Mutations in ZAP70 Leading to a SCID Phenotype with Normal Downstream In vitro Signaling;Journal of Clinical Immunology;2020-11-13
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