Human Severe Combined Immunodeficiency Due to a Defect in ZAP-70, a T Cell Tyrosine Kinase-Reference-Cited by-同舟云学术

Human Severe Combined Immunodeficiency Due to a Defect in ZAP-70, a T Cell Tyrosine Kinase

Published:1994-06-10 Issue:5165 Volume:264 Page:1596-1599
ISSN:0036-8075
Container-title:Science
language:en
Short-container-title:Science

Author:

Elder Melissa E.¹,Lin Dong¹,Clever Jared²,Chan Andrew C.³,Hope Thomas J.⁴,Weiss Arthur³,Parslow Tristram G.²

Affiliation:

1. Department of Pediatrics, University of California, San Francisco, CA 94143-0110, USA.

2. Department of Pathology and Department of Microbiology and Immunology, University of California, San Francisco, CA 94143, USA.

3. Howard Hughes Medical Institute, Department of Medicine and Department of Microbiology and Immunology, University of California, San Francisco, CA 94143, USA.

4. Infectious Diseases Laboratory, Salk Institute, San Diego, CA 92186, USA.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference30 articles.

1. APPLEBY, M.W., DEFECTIVE T-CELL RECEPTOR SIGNALING IN MICE LACKING THE THYMIC ISOFORM OF P59(FYN), CELL 70: 751 (1992).

2. CHAN, A.C., ZAP-70 - A 70 KD PROTEIN-TYROSINE KINASE THAT ASSOCIATES WITH THE TCR ZETA-CHAIN, CELL 71: 649 (1992).

3. CHAN, A.C., THE ZETA-CHAIN IS ASSOCIATED WITH A TYROSINE KINASE AND UPON T-CELL ANTIGEN RECEPTOR STIMULATION ASSOCIATES WITH ZAP-70, A 70-KDA TYROSINE PHOSPHOPROTEIN, PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA 88: 9166 (1991).

4. ZAP-70 Deficiency in an Autosomal Recessive form of Severe Combined Immunodeficiency

5. Thymic Selection of Cytotoxic T Cells Independent of CD8 α-Lck Association

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