RNA binding proteins and the pathological cascade in ALS/FTD neurodegeneration
Author:
Affiliation:
1. Department of Neurology, Keio University School of Medicine, Shinjuku-ku, Tokyo 160-8582, Japan.
2. Department of Pathology, Keio University School of Medicine, Shinjuku-ku, Tokyo 160-8582, Japan.
Abstract
Publisher
American Association for the Advancement of Science (AAAS)
Subject
General Medicine
Reference114 articles.
1. J. M. Charcot De la sclérose latérale amyotrophique - Symptomatologie Leçons sur les maladies du système nerveux faites à la Salpêtrière . (Delahaye 1880) vol. Tome II pp. 227–242.
2. Ubiquitinated TDP-43 in Frontotemporal Lobar Degeneration and Amyotrophic Lateral Sclerosis
3. TDP-43 is a component of ubiquitin-positive tau-negative inclusions in frontotemporal lobar degeneration and amyotrophic lateral sclerosis
4. Mutations in the FUS/TLS Gene on Chromosome 16 Cause Familial Amyotrophic Lateral Sclerosis
5. Mutations in FUS, an RNA Processing Protein, Cause Familial Amyotrophic Lateral Sclerosis Type 6
Cited by 70 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Targeting RACK1 to alleviate TDP-43 and FUS proteinopathy-mediated suppression of protein translation and neurodegeneration;Acta Neuropathologica Communications;2023-12-18
2. Drosha-dependent microRNAs modulate FUS-mediated neurodegeneration in vivo;Nucleic Acids Research;2023-10-04
3. Toward a high-resolution mechanism of intrinsically disordered protein self-assembly;The Journal of Biochemistry;2023-07-24
4. Globally reduced N6-methyladenosine (m6A) in C9ORF72-ALS/FTD dysregulates RNA metabolism and contributes to neurodegeneration;Nature Neuroscience;2023-06-26
5. A transient protein folding response targets aggregation in the early phase of TDP-43-mediated disease;2023-06-24
1.学者识别学者识别
2.学术分析学术分析
3.人才评估人才评估
"同舟云学术"是以全球学者为主线,采集、加工和组织学术论文而形成的新型学术文献查询和分析系统,可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容,当前同舟云学术共收录了国内外主流学术期刊6万余种,收集的期刊论文及会议论文总量共计约1.5亿篇,并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询!咨询电话:010-8811{复制后删除}0370
www.globalauthorid.com
TOP
Copyright © 2019-2024 北京同舟云网络信息技术有限公司 京公网安备11010802033243号 京ICP备18003416号-3