Targeting Tie2 and the host vascular response in sepsis-Reference-Cited by-同舟云学术

Targeting Tie2 and the host vascular response in sepsis

Published:2016-04-20 Issue:335 Volume:8 Page:
ISSN:1946-6234
Container-title:Science Translational Medicine
language:en
Short-container-title:Sci. Transl. Med.

Author:

Parikh Samir M.¹

Affiliation:

1. Center for Vascular Biology Research and Department of Medicine, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, MA 02215, USA.

Abstract

A new therapeutic approach to a complex and dire disease—sepsis—targets a signaling molecule in the vasculature (Han et al ., this issue).

Publisher

American Association for the Advancement of Science (AAAS)

Subject

General Medicine

Reference10 articles.

1. Amelioration of sepsis by TIE2 activation–induced vascular protection

2. Angiopoietin-2 may contribute to multiple organ dysfunction and death in sepsis*

3. Angiopoietin-1 Requires p190 RhoGAP to Protect against Vascular Leakage in Vivo

4. Excess Circulating Angiopoietin-2 May Contribute to Pulmonary Vascular Leak in Sepsis in Humans

5. Leakage-Resistant Blood Vessels in Mice Transgenically Overexpressing Angiopoietin-1

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