Comment on “Chronic Traumatic Encephalopathy in Blast-Exposed Military Veterans and a Blast Neurotrauma Mouse Model”-Reference-Cited by-同舟云学术

Comment on “Chronic Traumatic Encephalopathy in Blast-Exposed Military Veterans and a Blast Neurotrauma Mouse Model”

Published:2012-10-24 Issue:157 Volume:4 Page:
ISSN:1946-6234
Container-title:Science Translational Medicine
language:en
Short-container-title:Sci. Transl. Med.

Author:

Tisdall Martin¹²,Petzold Axel²³

Affiliation:

1. Department of Neurosurgery, The National Hospital for Neurology and Neurosurgery, London WC1N 3BG, UK.

2. Department of Neurocritical Care, The National Hospital for Neurology and Neurosurgery, London WC1N 3BG, UK.

3. Department of Neuroinflammation, UCL Institute of Neurology, London WC1N 1PJ, UK.

Abstract

In a case study, the authors report an increase in phosphorylated neurofilament heavy chain, a marker of neuroaxonal damage, in the plasma of a blast-exposed patient immediately after injury. They suggest that this phosphoprotein may be a useful body fluid indicator of acute blast traumatic brain injury.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

General Medicine

Reference5 articles.

1. Chronic Traumatic Encephalopathy in Blast-Exposed Military Veterans and a Blast Neurotrauma Mouse Model

2. Tau elevations in the brain extracellular space correlate with reduced amyloid-β levels and predict adverse clinical outcomes after severe traumatic brain injury.;Magnoni S.;Brain,2012

3. In vivo monitoring of neuronal loss in traumatic brain injury: A microdialysis study.;Petzold A.;Brain,2011

4. The phosphorylated axonal form of the neurofilament subunit NF-H (pNF-H) as a blood biomarker of traumatic brain injury.;Anderson K. J.;J. Neurotrauma,2008

5. Time-dependent changes in serum biomarker levels after blast traumatic brain injury.;Gyorgy A.;J. Neurotrauma,2011

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