Serum Amyloid P Inhibits Fibrosis Through FcγR-Dependent Monocyte-Macrophage Regulation in Vivo-Reference-Cited by-同舟云学术

Serum Amyloid P Inhibits Fibrosis Through FcγR-Dependent Monocyte-Macrophage Regulation in Vivo

Published:2009-11-04 Issue:5 Volume:1 Page:
ISSN:1946-6234
Container-title:Science Translational Medicine
language:en
Short-container-title:Sci. Transl. Med.

Author:

Castaño Ana P.¹²,Lin Shuei-Liong¹²³,Surowy Teresa⁴,Nowlin Brian T.¹,Turlapati Swathi A.¹,Patel Tejas²,Singh Ajay²,Li Shawn⁴,Lupher Mark L.⁴,Duffield Jeremy S.¹²

Affiliation:

1. Laboratory of Inflammation Research, Harvard Institutes of Medicine, 5th Floor, 4 Blackfan Circle, Boston, MA 02115, USA.

2. Renal Division, Department of Medicine, Brigham and Women’s Hospital and Harvard Medical School, Boston, MA 02115, USA.

3. National Taiwan University Hospital, College of Medicine, Taipei, Taiwan.

4. Promedior Inc., Malvern, PA 19355, USA.

Abstract

The fibrosis that occus in kidney after tissue injury can be inhibited by serum amyloid protein, which stimulates the cytokine interleukin 10.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

General Medicine

Reference49 articles.

1. The innate signaling of dangers and the dangers of innate signaling

2. Progression in Chronic Kidney Disease

3. Regulation of Fibrosis by the Immune System

4. The inflammatory macrophage: a story of Jekyll and Hyde

5. Alternative activation of macrophages

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