Reversal of Paralysis and Reduced Inflammation from Peripheral Administration of β-Amyloid in T H 1 and T H 17 Versions of Experimental Autoimmune Encephalomyelitis-Reference-Cited by-同舟云学术

Reversal of Paralysis and Reduced Inflammation from Peripheral Administration of β-Amyloid in T H 1 and T H 17 Versions of Experimental Autoimmune Encephalomyelitis

Published:2012-08 Issue:145 Volume:4 Page:
ISSN:1946-6234
Container-title:Science Translational Medicine
language:en
Short-container-title:Sci. Transl. Med.

Author:

Grant Jacqueline L.¹²,Ghosn Eliver Eid Bou³,Axtell Robert C.¹,Herges Katja¹,Kuipers Hedwich F.¹,Woodling Nathan S.¹²,Andreasson Katrin¹,Herzenberg Leonard A.³,Herzenberg Leonore A.³,Steinman Lawrence¹

Affiliation:

1. Department of Neurology and Neurological Sciences, Stanford University, Stanford, CA 94305, USA.

2. Neurosciences Graduate Program, School of Medicine, Stanford University, Stanford, CA 94305, USA.

3. Department of Genetics, Stanford University School of Medicine, Stanford, CA 94305, USA.

Abstract

β-Amyloid reduces inflammation and improves symptoms in a mouse model of multiple sclerosis.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

General Medicine

Reference60 articles.

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