Pain induces stable, active microcircuits in the somatosensory cortex that provide a therapeutic target-Reference-Cited by-同舟云学术

Pain induces stable, active microcircuits in the somatosensory cortex that provide a therapeutic target

Published:2021-03-19 Issue:12 Volume:7 Page:
ISSN:2375-2548
Container-title:Science Advances
language:en
Short-container-title:Sci. Adv.

Author:

Okada Takuya¹²^ORCID,Kato Daisuke³^ORCID,Nomura Yuki²^ORCID,Obata Norihiko²^ORCID,Quan Xiangyu⁴^ORCID,Morinaga Akihito¹³,Yano Hajime⁵,Guo Zhongtian¹³,Aoyama Yuki¹³,Tachibana Yoshihisa¹,Moorhouse Andrew J.⁶^ORCID,Matoba Osamu⁴^ORCID,Takiguchi Tetsuya⁵^ORCID,Mizobuchi Satoshi²,Wake Hiroaki¹³⁷^ORCID

Affiliation:

1. Division of System Neuroscience, Kobe University Graduate School of Medicine, Kobe, Japan.

2. Division of Anesthesiology, Kobe University Graduate School of Medicine, Kobe, Japan.

3. Department of Anatomy and Molecular Cell Biology, Nagoya University Graduate School of Medicine, Nagoya, Japan.

4. Department of System Science, Kobe University Graduate School of System Informatics, Kobe, Japan.

5. Department of Information Science, Kobe University Graduate School of System Informatics, Kobe, Japan.

6. School of Medical Sciences, UNSW Sydney, Australia.

7. Core Research for Evolutional Science and Technology, Japan Science and Technology Agency, Saitama, Japan.

Abstract

Upregulation of N-type Ca 2+ channel dependent subunits increases functional connections and synchronization for pain formation.

Funder

Japan Society for the Promotion of Science

Japan Science and Technology Agency

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference59 articles.

1. Relieving Pain in America: A Blueprint for Transforming Prevention, Care, Education, and Research

2. Transition to chronic pain: opportunities for novel therapeutics

3. Pain regulation by non-neuronal cells and inflammation

4. The Cloned Capsaicin Receptor Integrates Multiple Pain-Producing Stimuli

5. Impaired Nociception and Pain Sensation in Mice Lacking the Capsaicin Receptor

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