Type I interferons instigate fetal demise after Zika virus infection-Reference-Cited by-同舟云学术

Type I interferons instigate fetal demise after Zika virus infection

Published:2018-01-26 Issue:19 Volume:3 Page:
ISSN:2470-9468
Container-title:Science Immunology
language:en
Short-container-title:Sci. Immunol.

Author:

Yockey Laura J.¹^ORCID,Jurado Kellie A.¹,Arora Nitin²,Millet Alon¹^ORCID,Rakib Tasfia¹^ORCID,Milano Kristin M.³,Hastings Andrew K.⁴^ORCID,Fikrig Erol⁴⁵^ORCID,Kong Yong⁶^ORCID,Horvath Tamas L.⁷,Weatherbee Scott⁸,Kliman Harvey J.³^ORCID,Coyne Carolyn B.²⁹^ORCID,Iwasaki Akiko¹⁵^ORCID

Affiliation:

1. Department of Immunobiology, Yale University School of Medicine, New Haven, CT 06520, USA.

2. Department of Pediatrics, University of Pittsburgh School of Medicine, Pittsburgh, PA 15224, USA.

3. Department of Obstetrics, Gynecology, and Reproductive Sciences, Yale University School of Medicine, New Haven, CT 06520, USA.

4. Section of Infectious Diseases, Department of Internal Medicine, Yale University School of Medicine, New Haven, CT 06520, USA.

5. Howard Hughes Medical Institute, Chevy Chase, MD 20815, USA.

6. Yale School of Public Health, New Haven, CT 06520, USA.

7. Program in Integrative Cell Signaling and Neurobiology of Metabolism, Section of Comparative Medicine New Haven, CT 06520, USA.

8. Department of Genetics, Yale University School of Medicine, New Haven, CT 06520, USA.

9. Center for Microbial Pathogenesis, Children’s Hospital of Pittsburgh of UPMC (University of Pittsburgh Medical Center), Pittsburgh, PA 15224, USA.

Abstract

After Zika virus infection, fetal type I interferon signaling is responsible for abnormal placental development and fetal resorption.

Funder

National Institutes of Health

Howard Hughes Medical Institute

Publisher

American Association for the Advancement of Science (AAAS)

Subject

General Medicine,Immunology

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