TCR-stimulated changes in cell surface CD46 expression generate type 1 regulatory T cells

Author:

Ni Choileain Siobhan1,Hay Joanne1ORCID,Thomas Joelle2ORCID,Williams Anna3ORCID,Vermeren Matthieu M.1ORCID,Benezech Cecile4,Gomez-Salazar Mario1ORCID,Hugues Owen R.5ORCID,Vermeren Sonja1ORCID,Howie Sarah E. M.1ORCID,Dransfield Ian1ORCID,Astier Anne L.16ORCID

Affiliation:

1. Medical Research Council (MRC) Centre for Inflammation Research, University of Edinburgh, Queen’s Medical Research Institute, Edinburgh EH16 4TJ, UK.

2. Université Claude Bernard Lyon I, CNRS UMR 5310-INSERM U1217, F-69100 Lyon, France.

3. MRC Centre for Regenerative Medicine, University of Edinburgh, Edinburgh EH16 4UU, UK.

4. UK Centre for Cardiovascular Science, University of Edinburgh, Edinburgh EH16 4TJ, UK.

5. Millipore (U.K.) Limited, Croxley Green Business Park, Watford, Hertfordshire WD18 8ZB, UK.

6. Centre de Physiopathologie Toulouse-Purpan, INSERM U1043, CNRS U5282, Université de Toulouse, Toulouse F-31300, France.

Abstract

Defects in the T cell receptor–stimulated processing of a cell surface receptor may contribute to the pathogenesis of autoimmune diseases.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Cell Biology,Molecular Biology,Biochemistry

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