Use of Restriction Fragment Length Polymorphisms to Determine the Clonal Origin of Human Tumors

Author:

Vogelstein Bert1,Fearon Eric R.1,Hamilton Stanley R.2,Feinberg Andrew P.1

Affiliation:

1. Oncology Center, Johns Hopkins University School of Medicine, Baltimore, Maryland 21205

2. Department of Pathology, Johns Hopkins University School of Medicine and Johns Hopkins Hospital, Baltimore, Maryland 21205

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference42 articles.

1. ATKIN, N.B., CHROMOSOME ABNORMALITIES AS PRIMARY EVENTS IN HUMAN MALIGNANT DISEASE - EVIDENCE FROM MARKER CHROMOSOMES, JOURNAL OF THE NATIONAL CANCER INSTITUTE 36: 539 (1966).

2. BEUTLER, E, VALUE OF GENETIC VARIANTS OF GLUCOSE-6-PHOSPHATE DEHYDROGENASE IN TRACING ORIGIN OF MALIGNANT TUMORS, NEW ENGLAND JOURNAL OF MEDICINE 276: 389 (1967).

3. BIRD, A.P., USE OF RESTRICTION ENZYMES TO STUDY EUKARYOTIC DNA METHYLATION .1. METHYLATION PATTERN IN RIBOSOMAL DNA FROM XENOPUS-LAEVIS, JOURNAL OF MOLECULAR BIOLOGY 118: 27 (1978).

4. BORZY, M.S., FATAL LYMPHOMA AFTER TRANSPLANTATION OF CULTURED THYMUS IN CHILDREN WITH COMBINED IMMUNODEFICIENCY DISEASE, NEW ENGLAND JOURNAL OF MEDICINE 301: 565 (1979).

5. BOTSTEIN, D, CONSTRUCTION OF A GENETIC-LINKAGE MAP IN MAN USING RESTRICTION FRAGMENT LENGTH POLYMORPHISMS, AMERICAN JOURNAL OF HUMAN GENETICS 32: 314 (1980).

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