Comment on “Oxytocin-mediated GABA inhibition during delivery attenuates autism pathogenesis in rodent offspring”-Reference-Cited by-同舟云学术

Comment on “Oxytocin-mediated GABA inhibition during delivery attenuates autism pathogenesis in rodent offspring”

Published:2014-10-10 Issue:6206 Volume:346 Page:176-176
ISSN:0036-8075
Container-title:Science
language:en
Short-container-title:Science

Author:

Bambini-Junior Victorio¹²,Nunes Gustavo Della Flora¹²,Schneider Tomasz³,Gottfried Carmem¹²

Affiliation:

1. Federal University of Rio Grande do Sul, Research Group in Neuroglial Plasticity at the Department of Biochemistry, Institute of Health’s Basic Science, Porto Alegre, Rio Grande do Sul, Brazil.

2. Federal University of Rio Grande do Sul, Translational Research Group in Autism Spectrum Disorders (GETTEA), Porto Alegre, RS, Brazil.

3. School of Medicine, Pharmacy and Health, TS17 6BH, Durham University, Durham, UK.

Abstract

Tyzio et al . (Reports, 7 February 2014, p. 675) reported that bumetanide restored the impaired oxytocin-mediated γ-aminobutyric acid (GABA) excitatory-inhibitory shift during delivery in animal models of autism, ameliorating some autistic-like characteristics in the offspring. However, standard practices in the study of these models, such as the use of sex-dimorphic or males-only analyses and implementation of tests measuring social behavior, are lacking to definitely associate their findings to autism.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference15 articles.

1. The Emerging Biology of Autism Spectrum Disorders

2. Neocortical excitation/inhibition balance in information processing and social dysfunction

3. Oxytocin-Mediated GABA Inhibition During Delivery Attenuates Autism Pathogenesis in Rodent Offspring

4. A randomised controlled trial of bumetanide in the treatment of autism in children

5. Animal model of autism induced by prenatal exposure to valproate: Behavioral changes and liver parameters

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