VE-cadherin simultaneously stimulates and inhibits cell proliferation by altering cytoskeletal structure and tension
Author:
Affiliation:
1. Department of Biomedical Engineering, Johns Hopkins School of Medicine, 720 Rutland Avenue, Baltimore, MD 21205, USA
2. Department of Oncology, Johns Hopkins School of Medicine, 720 Rutland Avenue,Baltimore, MD 21205, USA
Abstract
Publisher
The Company of Biologists
Subject
Cell Biology
Link
http://journals.biologists.com/jcs/article-pdf/116/17/3571/1362296/3571.pdf
Reference46 articles.
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2. Assoian, R. K. and Schwartz, M. A. (2001). Coordinate signaling by integrins and receptor tyrosine kinases in the regulation of G1 phase cell-cycle progression. Curr. Opin. Genet. Dev.11,48-53.
3. Barth, A. I., Nathke, I. S. and Nelson, W. J.(1997). Cadherins, catenins and APC protein: interplay between cytoskeletal complexes and signaling pathways. Curr. Opin. Cell Biol.9,683-690.
4. Braga, V. (2000). Epithelial cell shape:cadherins and small GTPases. Exp. Cell Res.261, 83-90.
5. Breviario, F., Caveda, L., Corada, M., Martin-Padura, I.,Navarro, P., Golay, J., Introna, M., Gulino, D., Lampugnani, M. G. and Dejana,E. (1995). Functional properties of human vascular endothelial cadherin (7B4/Cadherin-5), an endothelium-specific cadherin. Arterioscler. Thromb. Vasc. Biol.15,1229-1239.
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