GIPC3 couples to MYO6 and PDZ domain proteins, and shapes the hair cell apical region
Author:
Affiliation:
1. Oregon Hearing Research Center & Vollum Institute, Oregon Health & Science University 1 , Portland, OR 97239 , USA
2. Stanford University 2 Department of Otolaryngology—Head & Neck Surgery , , Stanford, CA 94305 , USA
Abstract
Funder
National Institute on Deafness and Other Communication Disorders
Oregon Health & Science University
Publisher
The Company of Biologists
Subject
Cell Biology
Link
https://journals.biologists.com/jcs/article-pdf/doi/10.1242/jcs.261100/2795291/jcs261100.pdf
Reference56 articles.
1. The mouse Snell's waltzer deafness gene encodes an unconventional myosin;Avraham;Nat. Genet.,1995
2. Characterization of unconventional MYO6, the human homologue of the gene responsible for deafness in Snell's waltzer mice;Avraham;Hum. Mol. Genet.,1997
3. Biomolecular condensates: organizers of cellular biochemistry;Banani;Nat. Rev. Mol. Cell Biol.,2017
4. Myosin 18A coassembles with nonmuscle myosin 2 to form mixed bipolar filaments;Billington;Curr. Biol.,2015
5. Harnessing molecular motors for nanoscale pulldown in live cells;Bird;Mol. Biol. Cell,2017
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