Understanding gene-environment interactions in a mouse model of Crohn’s disease-Reference-Cited by-同舟云学术

Understanding gene-environment interactions in a mouse model of Crohn’s disease

Published:2011-01-01 Issue:1 Volume:4 Page:7-8
ISSN:1754-8411
Container-title:Disease Models & Mechanisms
language:en
Short-container-title:

Author:

Duckworth Carrie A.¹

Affiliation:

1. Department of Gastroenterology, Institute of Translational Medicine, University of Liverpool, Liverpool, L6933BX, UK e-mail: c.a.duckworth@liverpool.ac.uk

Publisher

The Company of Biologists

Subject

General Biochemistry, Genetics and Molecular Biology,Immunology and Microbiology (miscellaneous),Medicine (miscellaneous),Neuroscience (miscellaneous)

Link

http://journals.biologists.com/dmm/article-pdf/4/1/7/1619549/7.pdf

Reference11 articles.

1. Genome-wide association defines more than 30 distinct susceptibility loci for Crohn’s disease;Barrett;Nat. Genet.,2008

2. A key role for autophagy and the autophagy gene Atg16l1 in mouse and human intestinal Paneth cells;Cadwell;Nature,2008

3. A common role for Atg16L1, Atg5 and Atg7 in small intestinal Paneth cells and Crohn disease;Cadwell;Autophagy,2009

4. Virus-plus-susceptibility gene interaction determines Crohn’s disease gene Atg16L1 phenotypes in intestine;Cadwell;Cell,2010

5. Inflammatory bowel disease in a Swedish twin cohort: a long-term follow-up of concordance and clinical characteristics;Halfvarson;Gastroenterology,2003

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