Stabilization of neurotoxic Alzheimer amyloid- oligomers by protein engineering-Reference-Cited by-同舟云学术

Stabilization of neurotoxic Alzheimer amyloid- oligomers by protein engineering

Published:2010-08-16 Issue:35 Volume:107 Page:15595-15600
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Sandberg A.,Luheshi L. M.,Sollvander S.,Pereira de Barros T.,Macao B.,Knowles T. P. J.,Biverstal H.,Lendel C.,Ekholm-Petterson F.,Dubnovitsky A.,Lannfelt L.,Dobson C. M.,Hard T.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference32 articles.

1. Protein Misfolding, Functional Amyloid, and Human Disease

2. Amyloid β-Protein Assembly and Alzheimer Disease

3. Amyloid-β protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory

4. A specific amyloid-β protein assembly in the brain impairs memory

5. Aβ Oligomers Induce Neuronal Oxidative Stress through an N-Methyl-D-aspartate Receptor-dependent Mechanism That Is Blocked by the Alzheimer Drug Memantine

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