Renal phenotype in mice lacking the Kir5.1 (Kcnj16) K+ channel subunit contrasts with that observed in SeSAME/EAST syndrome-Reference-Cited by-同舟云学术

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Renal phenotype in mice lacking the Kir5.1 (Kcnj16) K+ channel subunit contrasts with that observed in SeSAME/EAST syndrome

Published:2011-06-01 Issue:25 Volume:108 Page:10361-10366
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Paulais M.,Bloch-Faure M.,Picard N.,Jacques T.,Ramakrishnan S. K.,Keck M.,Sohet F.,Eladari D.,Houillier P.,Lourdel S.,Teulon J.,Tucker S. J.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference40 articles.

1. RENAL K+ CHANNELS: Structure and Function

2. Impaired Renal NaCl Absorption in Mice Lacking the ROMK Potassium Channel, a Model for Type II Bartter's Syndrome

3. Proximal renal tubular acidosis in TASK2 K+ channel-deficient mice reveals a mechanism for stabilizing bicarbonate transport

4. Properties of an inwardly rectifying K+ channel in the basolateral membrane of mouse TAL

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