Impaired Renal NaCl Absorption in Mice Lacking the ROMK Potassium Channel, a Model for Type II Bartter's Syndrome-Reference-Cited by-同舟云学术

Impaired Renal NaCl Absorption in Mice Lacking the ROMK Potassium Channel, a Model for Type II Bartter's Syndrome

Published:2002-10 Issue:40 Volume:277 Page:37871-37880
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Lorenz John N.,Baird Nancy R.,Judd Louise M.,Noonan William T.,Andringa Anastasia,Doetschman Thomas,Manning Patrice A.,Liu Lynne H.,Miller Marian L.,Shull Gary E.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference43 articles.

1. Mutations in Na(K)Cl transporters in Gitelman's and Bartter's syndromes

2. Bartter's syndrome, hypokalaemic alkalosis with hypercalciuria, is caused by mutations in the Na–K–2CI cotransporter NKCC2

3. Genetic heterogeneity of Barter's syndrome revealed by mutations in the K+ channel, ROMK

4. Mutations in the chloride channel gene, CLCNKB, cause Bartter's syndrome type III

5. Mutation of BSND causes Bartter syndrome with sensorineural deafness and kidney failure

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