Antibodies to several conformation-dependent epitopes of gp120/gp41 inhibit CCR-5-dependent cell-to-cell fusion mediated by the native envelope glycoprotein of a primary macrophage-tropic HIV-1 isolate-Reference-Cited by-同舟云学术

Antibodies to several conformation-dependent epitopes of gp120/gp41 inhibit CCR-5-dependent cell-to-cell fusion mediated by the native envelope glycoprotein of a primary macrophage-tropic HIV-1 isolate

Published:1997-08-19 Issue:17 Volume:94 Page:9326-9331
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Verrier F. C.,Charneau P.,Altmeyer R.,Laurent S.,Borman A. M.,Girard M.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference46 articles.

1. CD4: Collaborator in immune recognition and HIV infection

2. Conformational changes induced in the human immunodeficiency virus envelope glycoprotein by soluble CD4 binding.

3. Dissociation of gp120 from HIV-1 Virions Induced by Soluble CD4

4. HIV-1 Entry Cofactor: Functional cDNA Cloning of a Seven-Transmembrane, G Protein-Coupled Receptor

5. A Dual-Tropic Primary HIV-1 Isolate That Uses Fusin and the β-Chemokine Receptors CKR-5, CKR-3, and CKR-2b as Fusion Cofactors

Cited by 38 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. A human monoclonal antibody blocks malaria transmission and defines a highly conserved neutralizing epitope on gametes;Nature Communications;2021-03-19

2. A high affinity human monoclonal antibody against Pfs230 binds multiple parasite stages and blocks oocyst formation in mosquitoes;2020-09-25

3. Reduction of HIV-1 Infectivity through Endoplasmic Reticulum-Associated Degradation-Mediated Env Depletion;Journal of Virology;2014-12-24

4. Neutralization sensitivity of HIV-1 subtype B’ clinical isolates from former plasma donors in China;Virology Journal;2013-01-05

5. Decidual soluble factors participate in the control of HIV-1 infection at the maternofetal interface;Retrovirology;2011-07-18