Toxic RNA as a driver of disease in a common form of ALS and dementia
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference11 articles.
1. Expanded GGGGCC Hexanucleotide Repeat in Noncoding Region of C9ORF72 Causes Chromosome 9p-Linked FTD and ALS
2. A Hexanucleotide Repeat Expansion in C9ORF72 Is the Cause of Chromosome 9p21-Linked ALS-FTD
3. A C9orf72 promoter repeat expansion in a Flanders-Belgian cohort with disorders of the frontotemporal lobar degeneration-amyotrophic lateral sclerosis spectrum: a gene identification study
4. Neurodegeneration the RNA way
5. Non-ATG–initiated translation directed by microsatellite expansions
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1. CBRPP: a new RNA-centric method to study RNA–protein interactions;RNA Biology;2021-02-17
2. A synthetic Pur-based peptide binds and alters G-quadruplex secondary structure present in the expanded RNA repeat of C9orf72 ALS/FTD;Biochimica et Biophysica Acta (BBA) - Molecular Cell Research;2020-06
3. A Peptidylic Inhibitor for Neutralizing (GGGGCC)-Associated Neurodegeneration in C9ALS-FTD;Molecular Therapy - Nucleic Acids;2019-06
4. Cellular and circuit mechanisms underlying spinocerebellar ataxias;The Journal of Physiology;2016-06-12
5. A retrospective review of the progress in amyotrophic lateral sclerosis drug discovery over the last decade and a look at the latest strategies;Expert Opinion on Drug Discovery;2015-08-26
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