Continuous fat oxidation in acetyl CoA carboxylase 2 knockout mice increases total energy expenditure, reduces fat mass, and improves insulin sensitivity-Reference-Cited by-同舟云学术

Continuous fat oxidation in acetyl CoA carboxylase 2 knockout mice increases total energy expenditure, reduces fat mass, and improves insulin sensitivity

Published:2007-10-08 Issue:42 Volume:104 Page:16480-16485
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Choi C. S.,Savage D. B.,Abu-Elheiga L.,Liu Z.-X.,Kim S.,Kulkarni A.,Distefano A.,Hwang Y.-J.,Reznick R. M.,Codella R.,Zhang D.,Cline G. W.,Wakil S. J.,Shulman G. I.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference47 articles.

1. FATTY ACID SYNTHESIS AND ITS REGULATION

2. The regulation of Acetyl-CoA carboxylase—a potential target for the action of hypolipidemic agents

3. Regulation of mammalian acetyl-CoA carboxylase

4. The Mitochondrial Carnitine Palmitoyltransferase System - From Concept to Molecular Analysis

5. Continuous Fatty Acid Oxidation and Reduced Fat Storage in Mice Lacking Acetyl-CoA Carboxylase 2

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