No reason to reconsider HIF-2 as an oncogene in neuroblastoma and other cancer forms-Reference-Cited by-同舟云学术

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No reason to reconsider HIF-2 as an oncogene in neuroblastoma and other cancer forms

Published:2017-12-12 Issue:51 Volume:114 Page:E10856-E10858
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proc Natl Acad Sci USA

Author:

Mohlin Sofie,von Stedingk Kristoffer,Pietras Alexander,Påhlman Sven

Funder

Cancerfonden

Barncancerfonden

Ragnar Söderbergs stiftelse

Svenska Forskningsrådet Formas

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference10 articles.

1. Combined epigenetic and differentiation-based treatment inhibits neuroblastoma tumor growth and links HIF2α to tumor suppression

2. Hypoxia-Inducible Factors in Physiology and Medicine

3. Oxygen Sensing by Metazoans: The Central Role of the HIF Hydroxylase Pathway

4. The Expression and Distribution of the Hypoxia-Inducible Factors HIF-1α and HIF-2α in Normal Human Tissues, Cancers, and Tumor-Associated Macrophages

5. Spatiotemporal Dynamics of Intratumoral Immune Cells Reveal the Immune Landscape in Human Cancer

Cited by 6 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Structural basis for binding of the renal carcinoma target hypoxia‐inducible factor 2α to prolyl hydroxylase domain 2;Proteins: Structure, Function, and Bioinformatics;2023-07-14

2. DYRK3 contributes to differentiation and hypoxic control in neuroblastoma;Biochemical and Biophysical Research Communications;2021-08

3. Oxygen-sensing mechanisms across eukaryotic kingdoms and their roles in complex multicellularity;Science;2020-10-23

4. Targeting the HIF2–VEGF axis in renal cell carcinoma;Nature Medicine;2020-10

5. Deletion of an intronic HIF-2α binding site suppresses hypoxia-induced WT1 expression;Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms;2019-01

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