Human and mouse homologs of Escherichia coli DinB (DNA polymerase IV), members of the UmuC/DinB superfamily
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference36 articles.
1. The Mutagenesis Proteins UmuD′ and UmuC Prevent Lethal Frameshifts While Increasing Base Substitution Mutations
2. Biochemical basis of SOS-induced mutagenesis in Escherichia coli: Reconstitution of in vitro lesion bypass dependent on the UmuD'2C mutagenic complex and RecA protein
3. dinP, a new gene in Escherichia coli, whose product shows similarities to UmuC and its homologues
4. Role of RecA protein in untargeted UV mutagenesis of bacteriophage lambda: evidence for the requirement for the dinB gene.
5. Multiple pathways for SOS-induced mutagenesis in Escherichia coli: An overexpression of dinB/dinP results in strongly enhancing mutagenesis in the absence of any exogenous treatment to damage DNA
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