Targeted disruption of an erythrocyte binding antigen in Plasmodium falciparum is associated with a switch toward a sialic acid-independent pathway of invasion-Reference-Cited by-同舟云学术

Targeted disruption of an erythrocyte binding antigen in Plasmodium falciparum is associated with a switch toward a sialic acid-independent pathway of invasion

Published:2000-06-20 Issue:13 Volume:97 Page:7509-7514
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Reed M. B.,Caruana S. R.,Batchelor A. H.,Thompson J. K.,Crabb B. S.,Cowman A. F.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference32 articles.

1. Transformation of Plasmodium falciparum malaria parasites by homologous integration of plasmids that confer resistance to pyrimethamine.

2. Characterization of promoters and stable transfection by homologous and nonhomologous recombination in Plasmodium falciparum.

3. Targeted Gene Disruption Shows That Knobs Enable Malaria-Infected Red Cells to Cytoadhere under Physiological Shear Stress

4. Glycophorin B as an EBA-175 independent Plasmodium falciparum receptor of human erythrocytes

5. Susceptibility to invasion by Plasmodium falciparum of some human erythrocytes carrying rare blood group antigens

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