Selenoprotein N is required for ryanodine receptor calcium release channel activity in human and zebrafish muscle-Reference-Cited by-同舟云学术

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Selenoprotein N is required for ryanodine receptor calcium release channel activity in human and zebrafish muscle

Published:2008-08-19 Issue:34 Volume:105 Page:12485-12490
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Jurynec M. J.,Xia R.,Mackrill J. J.,Gunther D.,Crawford T.,Flanigan K. M.,Abramson J. J.,Howard M. T.,Grunwald D. J.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference39 articles.

1. Congenital myopathies and congenital muscular dystrophies

2. Understanding the importance of selenium and selenoproteins in muscle function

3. Molecular mechanisms and phenotypic variation in RYR1-related congenital myopathies

4. Animal models of human disease: zebrafish swim into view

5. SEPN1: Associated with congenital fiber-type disproportion and insulin resistance

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2. Endoplasmic reticulum-resident selenoproteins and their roles in glucose and lipid metabolic disorders;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2024-08

3. SEPN1-related myopathy depends on the oxidoreductase ERO1A and is druggable with the chemical chaperone TUDCA;Cell Reports Medicine;2024-03

4. The beneficial and toxic effects of selenium on zebrafish. A systematic review of the literature;Toxicology Research;2024-03-01

5. Zebrafish and cellular models ofSELENON-Related Myopathy exhibit novel embryonic and metabolic phenotypes;2024-02-26

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