Vasculature remodeling by pressure, caveolae, calcium, and kinases-Reference-Cited by-同舟云学术

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Vasculature remodeling by pressure, caveolae, calcium, and kinases

Published:2022-05-18 Issue:21 Volume:119 Page:
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proc. Natl. Acad. Sci. U.S.A.

Author:

Marx Abigail M.¹,Marx Steven O.²³^ORCID

Affiliation:

1. University of Michigan, Ann Arbor, MI 48109

2. Division of Cardiology, Department of Medicine, Vagelos College of Physicians and Surgeons, Columbia University, New York, NY 10032

3. Department of Pharmacology and Molecular Signaling, Vagelos College of Physicians and Surgeons, Columbia University, New York, NY 10032

Funder

HHS | NIH | National Heart, Lung, and Blood Institute

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Link

https://pnas.org/doi/pdf/10.1073/pnas.2204968119

Reference19 articles.

1. Smooth Muscle Cell Changes During Flow-Related Remodeling of Rat Mesenteric Resistance Arteries

2. Blood flow-dependent arterial remodelling is facilitated by inflammation but directed by vascular tone

3. High Pressure Promotes Monocyte Adhesion to the Vascular Wall

4. A molecular complex of Ca _v 1.2/CaMKK2/CaMK1a in caveolae is responsible for vascular remodeling via excitation–transcription coupling

5. Myogenic responses are independent of the endothelium in rat pressurized posterior cerebral arteries;McCarron J. G.;Blood Vessels,1989

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