Molecular mechanisms for the subversion of MyD88 signaling by TcpC from virulent uropathogenic Escherichia coli-Reference-Cited by-同舟云学术

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Molecular mechanisms for the subversion of MyD88 signaling by TcpC from virulent uropathogenic Escherichia coli

Published:2013-04-08 Issue:17 Volume:110 Page:6985-6990
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Snyder G. A.,Cirl C.,Jiang J.,Chen K.,Waldhuber A.,Smith P.,Rommler F.,Snyder N.,Fresquez T.,Durr S.,Tjandra N.,Miethke T.,Xiao T. S.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference56 articles.

1. Innate Immune Recognition

2. Toll-like receptor signalling

3. The family of five: TIR-domain-containing adaptors in Toll-like receptor signalling

4. Unresponsiveness of MyD88-Deficient Mice to Endotoxin

5. Targeted Disruption of the MyD88 Gene Results in Loss of IL-1- and IL-18-Mediated Function

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4. The metabolic cofactor Coenzyme A enhances alternative macrophage activation via MyD88-linked signaling;2024-03-31

5. Purpurolide C-based microneedle promotes macrophage-mediated diabetic wound healing via inhibiting TLR4-MD2 dimerization and MYD88 phosphorylation;Acta Pharmaceutica Sinica B;2023-12

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