Bisphenol A Exposure during Adulthood Causes Augmentation of Follicular Atresia and Luteal Regression by Decreasing 17β-Estradiol Synthesis via Downregulation of Aromatase in Rat Ovary-Reference-Cited by-同舟云学术

Bisphenol A Exposure during Adulthood Causes Augmentation of Follicular Atresia and Luteal Regression by Decreasing 17β-Estradiol Synthesis via Downregulation of Aromatase in Rat Ovary

Published:2013-06 Issue:6 Volume:121 Page:663-669
ISSN:0091-6765
Container-title:Environmental Health Perspectives
language:en
Short-container-title:Environmental Health Perspectives

Author:

Lee Seung Gee¹²,Kim Ji Young¹³,Chung Jin-Yong¹³,Kim Yoon-Jae¹²,Park Ji-Eun¹²,Oh Seunghoon⁴,Yoon Yong-Dal⁵,Yoo Ki Soo¹²,Yoo Young Hyun¹²³,Kim Jong-Min¹²³

Affiliation:

1. Department of Anatomy and Cell Biology, College of Medicine, Dong-A University, Busan, Korea

2. Mitochondria Hub Regulation Center, and

3. Medical Science Research Center, Dong-A University, Busan, Korea

4. Department of Physiology, College of Medicine, Dankook University, Cheonan, Korea

5. Department of Life Science, College of Natural Sciences, Hanyang University, Seoul, Korea

Publisher

Environmental Health Perspectives

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health

Reference39 articles.

1. Neonatal Bisphenol-A Exposure Alters Rat Reproductive Development and Ovarian Morphology Without Impairing Activation of Gonadotropin-Releasing Hormone Neurons1

2. Estrogens inhibit and androgens enhance ovarian granulosa cell apoptosis.

3. Caspase-3 in the Rat Ovary: Localization and Possible Role in Follicular Atresia and Luteal Regression1

4. Perinatal Exposure to Environmentally Relevant Levels of Bisphenol A Decreases Fertility and Fecundity in CD-1 Mice

5. Exposure of the U.S. Population to Bisphenol A and 4- tertiary -Octylphenol: 2003–2004

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