Activation of Both the Calpain and Ubiquitin-Proteasome Systems Contributes to Septic Cardiomyopathy through Dystrophin Loss/Disruption and mTOR Inhibition
Author:
Funder
Fundação de Amparo à Pesquisa do Estado de São Paulo
Conselho Nacional de Desenvolvimento Científico e Tecnológico
Publisher
Public Library of Science (PLoS)
Subject
Multidisciplinary
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3. Myocardial structural changes in long-term human severe sepsis/septic shock may be responsible for cardiac dysfunction;MA Rossi;Shock,2007
4. Disruption of sarcolemmal dystrophin and beta-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis;MRN Celes;Lab Invest,2010
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3. Calpains Released from Necrotic Tumor Cells Enhance Antigen Cross-Presentation to Activate CD8+ T Cells In Vitro;The Journal of Immunology;2022-11-01
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