The FYVE Domain of Smad Anchor for Receptor Activation (SARA) Is Required to Prevent Skin Carcinogenesis, but Not in Mouse Development-Reference-Cited by-同舟云学术

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The FYVE Domain of Smad Anchor for Receptor Activation (SARA) Is Required to Prevent Skin Carcinogenesis, but Not in Mouse Development

Published:2014-08-29 Issue:8 Volume:9 Page:e105299
ISSN:1932-6203
Container-title:PLoS ONE
language:en
Short-container-title:PLoS ONE

Author:

Chang Huang-Ming,Lin Yu-Ying,Tsai Pei-Chun,Liang Chung-Tiang,Yan Yu-Ting

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference51 articles.

1. Regulation of differentiation by TGF-beta;HL Moses;Curr Opin Genet Dev,1996

2. Mechanisms of TGF-beta signaling in regulation of cell growth and differentiation;A Moustakas;Immunol Lett,2002

3. TGF-beta and epithelial-to-mesenchymal transitions;J Zavadil;Oncogene,2005

4. The differential role of Smad2 and Smad3 in the regulation of pro-fibrotic TGFbeta1 responses in human proximal-tubule epithelial cells;MK Phanish;Biochem J,2006

5. TGF-beta signaling in tumor suppression and cancer progression;R Derynck;Nat Genet,2001

Cited by 3 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Fine-Tuning the TGFβ Signaling Pathway by SARA During Neuronal Development;Frontiers in Cell and Developmental Biology;2020-09-03

2. New Player in Endosomal Trafficking: Differential Roles of Smad Anchor for Receptor Activation (SARA) Protein;Molecular and Cellular Biology;2018-12-15

3. Involvement of SARA in Axon and Dendrite Growth;PLOS ONE;2015-09-25

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