The CaM Kinase CMK-1 Mediates a Negative Feedback Mechanism Coupling the C. elegans Glutamate Receptor GLR-1 with Its Own Transcription-Reference-Cited by-同舟云学术

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The CaM Kinase CMK-1 Mediates a Negative Feedback Mechanism Coupling the C. elegans Glutamate Receptor GLR-1 with Its Own Transcription

Published:2016-07-27 Issue:7 Volume:12 Page:e1006180
ISSN:1553-7404
Container-title:PLOS Genetics
language:en
Short-container-title:PLoS Genet

Author:

Moss Benjamin J.,Park Lidia,Dahlberg Caroline L.,Juo Peter

Funder

National Institute of Neurological Disorders and Stroke

National Center for Research Resources

National Institute of General Medical Sciences

Publisher

Public Library of Science (PLoS)

Subject

Cancer Research,Genetics (clinical),Genetics,Molecular Biology,Ecology, Evolution, Behavior and Systematics

Reference61 articles.

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2. The self-tuning neuron: synaptic scaling of excitatory synapses;G.G. Turrigiano;Cell,2008

3. Adaptation to synaptic inactivity in hippocampal neurons;T.C. Thiagarajan;Neuron,2005

4. Activity-dependent scaling of quantal amplitude in neocortical neurons;G.G. Turrigiano;Nature,1998

5. Activity-dependent modulation of synaptic AMPA receptor accumulation;R.J. O'Brien;Neuron,1998

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3. Ca²⁺/CaM binding to CaMKI promotes IMA-3 importin binding and nuclear translocation in sensory neurons to control behavioral adaptation;ELIFE;2021

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