Advanced Glycation Endproducts and Bone Material Properties in Type 1 Diabetic Mice-Reference-Cited by-同舟云学术

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Advanced Glycation Endproducts and Bone Material Properties in Type 1 Diabetic Mice

Published:2016-05-03 Issue:5 Volume:11 Page:e0154700
ISSN:1932-6203
Container-title:PLOS ONE
language:en
Short-container-title:PLoS ONE

Author:

Rubin Mishaela R.,Paschalis Eleftherios P.,Poundarik Atharva,Sroga Gyna E.,McMahon Donald J.,Gamsjaeger Sonja,Klaushofer Klaus,Vashishth Deepak

Funder

Columbia University Irving Institute for Clinical and Translational Research

National Institutes of Health

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference56 articles.

1. Discrepancies in bone mineral density and fracture risk in patients with type 1 and type 2 diabetes—a meta-analysis;P Vestergaard;Osteoporos Int,2007

2. Accelerated age-related browning of human collagen in diabetes mellitus;VM Monnier;Proceedings of the National Academy of Sciences of the United States of America,1984

3. The role of the collagen matrix in skeletal fragility;D Vashishth;Current osteoporosis reports,2007

4. Role of the Maillard reaction in aging of tissue proteins. Advanced glycation end product-dependent increase in imidazolium cross-links in human lens proteins;EB Frye;The Journal of biological chemistry,1998

5. Glycation products as markers and predictors of the progression of diabetic complications;VM Monnier;Annals of the New York Academy of Sciences,2005

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1. Bone and muscle differences in children and adolescents with type 1 diabetes: The mediating role of physical activity;Bone;2024-10

2. Role of advanced glycation endproducts in bone fragility in type 1 diabetes;Bone;2024-01

3. Advanced-Glycation Endproducts: How cross-linking properties affect the collagen fibril behavior;Journal of the Mechanical Behavior of Biomedical Materials;2023-12

4. Advanced glycation and glycoxidation end products in bone;Bone;2023-11

5. Accumulation of advanced glycation end products promotes atrophic nonunion incidence in mice through a CtBP1/2-dependent mechanism;Experimental Cell Research;2023-11

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