Folding Landscape of Mutant Huntingtin Exon1: Diffusible Multimers, Oligomers and Fibrils, and No Detectable Monomer-Reference-Cited by-同舟云学术

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Folding Landscape of Mutant Huntingtin Exon1: Diffusible Multimers, Oligomers and Fibrils, and No Detectable Monomer

Published:2016-06-06 Issue:6 Volume:11 Page:e0155747
ISSN:1932-6203
Container-title:PLOS ONE
language:en
Short-container-title:PLoS ONE

Author:

Sahoo Bankanidhi,Arduini Irene,Drombosky Kenneth W.,Kodali Ravindra,Sanders Laurie H.,Greenamyre J. Timothy,Wetzel Ronald

Funder

National Institute on Aging

National Institute of Environmental Health Sciences

Hereditary Disease Foundation

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference70 articles.

1. Huntington's disease: underlying molecular mechanisms and emerging concepts;J Labbadia;Trends Biochem Sci,2013

2. Huntington's Disease;G Bates,2014

3. Huntington disease;GP Bates;Nature Reviews Disease Primers,2015

4. Comparative study of naturally occurring huntingtin fragments in Drosophila points to exon 1 as the most pathogenic species in Huntington's disease;BA Barbaro;Hum Mol Genet,2015

5. Huntington's Disease;R Wetzel,2014

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1. A Targetable Self-association Surface of the Huntingtin exon1 Helical Tetramer Required for Assembly of Amyloid Pre-nucleation Oligomers;Journal of Molecular Biology;2024-06

2. Nucleation of Huntingtin Aggregation Proceeds via Conformational Conversion of Pre‐Formed, Sparsely‐Populated Tetramers;Advanced Science;2024-03-12

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4. A clinical study and future prospects for bioactive compounds and semi-synthetic molecules in the therapies for Huntington's disease;Molecular Neurobiology;2023-09-12

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