Unusual Structures Are Present in DNA Fragments Containing Super-Long Huntingtin CAG Repeats-Reference-Cited by-同舟云学术

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Unusual Structures Are Present in DNA Fragments Containing Super-Long Huntingtin CAG Repeats

Published:2011-02-11 Issue:2 Volume:6 Page:e17119
ISSN:1932-6203
Container-title:PLoS ONE
language:en
Short-container-title:PLoS ONE

Author:

Duzdevich Daniel,Li Jinliang,Whang Jhoon,Takahashi Hirohide,Takeyasu Kunio,Dryden David T. F.,Morton A. Jennifer,Edwardson J. Michael

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference53 articles.

1. Trinucleotide repeat disorders.;HT Orr;Annu Rev Neurosci,2007

2. Repeat instability as the basis for human diseases and as a potential target for therapy.;A López Castel;Nat Rev Mol Cell Biol,2010

3. Venezuelan kindreds reveal that genetic and environmental factors modulate Huntington's disease age of onset.;NS Wexler;Proc Natl Acad Sci USA,2004

4. Genome-wide significance for a modifier of age at neurological onset in Huntington's disease at 6q23-24: the HD MAPS study.;JL Li;BMC Med Genet,2006

5. The relationship between CAG repeat length and age of onset differs for Huntington's disease patients with juvenile onset or adult onset.;JM Andresen;Ann Hum Genet,2007

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3. Subcellular Localization And Formation Of Huntingtin Aggregates Correlates With Symptom Onset And Progression In A Huntington’S Disease Model;Brain Communications;2020

4. Experimenting with Trinucleotide Repeats: Facts and Technical Issues;Methods in Molecular Biology;2019-10-05

5. Antagonistic pleiotropy in mice carrying a CAG repeat expansion in the range causing Huntington’s disease;Scientific Reports;2019-01-10

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