Haploinsufficiency for Translation Elongation Factor eEF1A2 in Aged Mouse Muscle and Neurons Is Compatible with Normal Function-Reference-Cited by-同舟云学术

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Haploinsufficiency for Translation Elongation Factor eEF1A2 in Aged Mouse Muscle and Neurons Is Compatible with Normal Function

Published:2012-07-25 Issue:7 Volume:7 Page:e41917
ISSN:1932-6203
Container-title:PLoS ONE
language:en
Short-container-title:PLoS ONE

Author:

Griffiths Lowri A.,Doig Jennifer,Churchhouse Antonia M. D.,Davies Faith C. J.,Squires Charlotte E.,Newbery Helen J.,Abbott Catherine M.

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference21 articles.

1. Ultrastructural, morphometric, and immunocytochemical study of anterior horn cells in mice with “wasted” mutation.;HL Lutsep;J Neuropathol Exp Neurol,1989

2. Progressive loss of motor neuron function in wasted mice: effects of a spontaneous null mutation in the gene for the eEF1 A2 translation factor.;HJ Newbery;J Neuropathol Exp Neurol,2005

3. 'Wasted', a new mutant of the mouse with abnormalities characteristic to ataxia telangiectasia.;LD Shultz;Nature,1982

4. The lethal mutation of the mouse wasted (wst) is a deletion that abolishes expression of a tissue-specific isoform of translation elongation factor 1alpha, encoded by the Eef1a2 gene.;DM Chambers;Proc Natl Acad Sci U S A,1998

5. Characterization of the statin-like S1 and rat elongation factor 1 alpha as two distinctly expressed messages in rat.;DK Ann;J Biol Chem,1992

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