In vivo glucoregulation and tissue-specific glucose uptake in female Akt substrate 160 kDa knockout rats
Author:
Funder
National Institutes of Health
Diabetes Research Center
Publisher
Public Library of Science (PLoS)
Subject
Multidisciplinary
Reference50 articles.
1. Roles of TBC1D1 and TBC1D4 in insulin- and exercise-stimulated glucose transport of skeletal muscle;GD Cartee;Diabetologia,2015
2. Insulin-stimulated phosphorylation of a Rab GTPase-activating protein regulates GLUT4 translocation;H Sano;J Biol Chem,2003
3. Deletion of Rab GAP AS160 modifies glucose uptake and GLUT4 translocation in primary skeletal muscles and adipocytes and impairs glucose homeostasis;MN Lansey;Am J Physiol Endocrinol Metab,2012
4. AS160 deficiency causes whole-body insulin resistance via composite effects in multiple tissues;HY Wang;Biochem J,2013
5. Whole body glucoregulation and tissue-specific glucose uptake in a novel Akt substrate of 160 kDa knockout rat model;EB Arias;PLoS One,2019
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1. Phosphorylation of AS160-serine 704 is not essential for exercise-increase in insulin-stimulated glucose uptake by skeletal muscles from female or male rats;American Journal of Physiology-Endocrinology and Metabolism;2024-06-01
2. AS160 expression, but not AS160 Serine‐588, Threonine‐642, and Serine‐704 phosphorylation, is essential for elevated insulin‐stimulated glucose uptake by skeletal muscle from female rats after acute exercise;The FASEB Journal;2023-06-08
3. Akt substrate of 160 kDa is essential for the calorie restriction-induced increase in insulin-stimulated glucose uptake by skeletal muscle of female rats;Applied Physiology, Nutrition, and Metabolism;2023-03-01
4. Deletion of Tbc1d4/As160 abrogates cardiac glucose uptake and increases myocardial damage after ischemia/reperfusion;Cardiovascular Diabetology;2023-01-27
5. Epiregulin as an Alternative Ligand for Leptin Receptor Alleviates Glucose Intolerance without Change in Obesity;Cells;2022-01-26
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