PEP-1-paraoxonase 1 fusion protein prevents cytokine-induced cell destruction and impaired insulin secretion in rat insulinoma cells-Reference-Cited by-同舟云学术

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PEP-1-paraoxonase 1 fusion protein prevents cytokine-induced cell destruction and impaired insulin secretion in rat insulinoma cells

Published:2018-10-31 Issue:10 Volume:51 Page:538-543
ISSN:1976-670X
Container-title:BMB Reports
language:en
Short-container-title:BMB Rep.

Author:

Lee Su Jin¹,Kang Hyung Kyung²,Choi Yeon Joo³,Eum Won Sik³,Park Jinseu³,Choi Soo Young³,Kwon Hyeok Yil¹

Affiliation:

1. Department of Physiology, College of Medicine, Hallym University, Chuncheon 24252, Korea

2. Department of Physiology, College of Medicine, Korea University, Seoul 02841, Korea

3. Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon 24252, Korea

Funder

National Research Foundation of Korea(NRF)

Ministry of Education(Ministry of Education of the Republic of Korea)

Publisher

Korean Society for Biochemistry and Molecular Biology - BMB Reports

Subject

Molecular Biology,Biochemistry,General Medicine

Link

http://pdf.medrang.co.kr/BMB/2018/051/BMB051-10-11.pdf

Reference27 articles.

1. A choice of death - the signal-transduction of immune-mediated beta-cell apoptosis

2. The role of reactive oxygen species and proinflammatory cytokines in type 1 diabetes pathogenesis

3. Nitric oxide contributes to cytokine-induced apoptosis in pancreatic beta cells via potentiation of JNK activity and inhibition of Akt

4. Proinflammatory Cytokines Activate the Intrinsic Apoptotic Pathway in β-Cells

5. Macrophage-foam cell formation in streptozotocin-induced diabetic mice: Stimulatory effect of glucose

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