The role of reactive oxygen species and proinflammatory cytokines in type 1 diabetes pathogenesis

Author:

Padgett Lindsey E.1,Broniowska Katarzyna A.2,Hansen Polly A.2,Corbett John A.2,Tse Hubert M.1

Affiliation:

1. Department of Microbiology, Comprehensive Diabetes Center University of Alabama at Birmingham Birmingham Alabama

2. Department of Biochemistry The Medical College of Wisconsin Milwaukee Wisconsin

Publisher

Wiley

Subject

History and Philosophy of Science,General Biochemistry, Genetics and Molecular Biology,General Neuroscience

Reference195 articles.

1. Type 1 Diabetes: Etiology, Immunology, and Therapeutic Strategies

2. Oxidative Stress in Type 1 Diabetes

3. Inflammatory cytokines provide a third signal for activation of naive CD4+ and CD8+ T cells;Curtsinger J.M.;J. Immunol,1999

4. Inflammatory cytokines enhance the in vivo clonal expansion and differentiation of antigen‐activated CD4+ T cells;Pape K.A.;J. Immunol,1997

5. Mechanistic analysis of the immunomodulatory effects of a catalytic antioxidant on antigen-presenting cells: implication for their use in targeting oxidation–reduction reactions in innate immunity

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