Protective effects of N,4,5-trimethylthiazol-2-amine hydrochloride on hypoxia-induced β-amyloid production in SH-SY5Y cells
Author:
Affiliation:
1. Department of Biochemistry and Molecular Biology, University of Ulsan College of Medicine, Seoul 05505, Korea
2. Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon 24252, Korea
Funder
Asan Institute for Life Sciences, Asan Medical Center
Publisher
Korean Society for Biochemistry and Molecular Biology - BMB Reports
Subject
Molecular Biology,Biochemistry,General Medicine
Link
http://pdf.medrang.co.kr/BMB/2019/052/BMB052-07-04.pdf
Reference30 articles.
1. Neuroprotective effect of steroidal alkaloids on glutamate-induced toxicity by preserving mitochondrial membrane potential and reducing oxidative stress
2. Effect of Hypoxia/Ischemia and Hypoxic Preconditioning/Reperfusion on Expression of Some Amyloid-Degrading Enzymes
3. Brain Ischemia Activates β- and γ-Secretase Cleavage of Amyloid Precursor Protein: Significance in Sporadic Alzheimer’s Disease
4. Hypoxia/ischemia activate processing of Amyloid Precursor Protein: impact of vascular dysfunction in the pathogenesis of Alzheimer's disease
5. Chronic Intermittent Hypoxia/Reoxygenation Facilitate Amyloid-β Generation in Mice
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